![]() qu'une efférence des NSC. Du fait de la présence de récepteurs melatoniner- giques dans les SCN, la MLT exogène a des propriétés chronobiotiques. Les ago- nistes et antagonistes spécifiques aux di- vers récepteurs de la MLT sont donc des outils pharmacologiques utiles aux inno- vations thérapeutiques dans ce domaine particulier. 1. nophotic stimuli to synchronize the mammalian circadian clock. Frontiers in Bioscience 2003;8:246-57. of food anticipation. Europ. J. Neurosc 2009;30:1676- 87. circadian clock? Med Sci (Paris) 2005;21:66-72. oscillators in mammals: time and food. J Biol Rhythms 2003;18:250-60. cerebellum harbors a circadian oscillator involved in food anticipation J. Neuroscience 2010;30:1894-1904. and internal time-giver in the circadian clocks network. J Physiol Paris 2011 (in press). real-time reporting of circadian dynamics reveals persistent circadian oscillations in mouse peripheral tissues. Proc Natl Acad Sci USA 2004;101:5339-46. calorie restriction to control daily timing in mammals. J Comp Physiol B 2010;180:631-44. mammalian circadian timing system. J Biol Rhythms 2009;24:3-15. coupling signal from the transplanted suprachiasmatic nucleus controlling circadian locomotor rhythms. Nature 1996;382:810-13. transmits the behavioural circadian rhythm of the suprachiasmatic nucleus. Nature 2002;417:405-10. central and peripheral clocks. Nat Rev Neurosci 2001;7:521-6. circadian rhythms: the efferent projections of the suprachiasmatic nucleus. Prog Brain Res 1996;111:229- 40. mammalian suprachiasmatic nucleus: coding circadian time by transmitter selection and specific targeting. Cell Tissue Res 2002;309:109-18. the hypothalamic balance of life. J Biol Rhythms 2006;21:458-69. output mechanisms. Essays Biochem 2011;49:137-51. Endocrinol 2009;200:3-22. environment induced inhibition of corticosterone secretion: physiological evidence for a suprachiasmatic nucleus mediated neuronal hypothalamo-adrenal cortex pathway. Brain Res 1997;758:229-36. driving influence of the suprachiasmatic nucleus on adrenal sensitivity to AC TH. Am J Physiol Endocrinol Metab 2002;282:E458-465. tryptophan hydroxylase-2-mRNA: within raphe neurons is induced by corticoid daily surge and modulated by enhanced mediated nycthemeral and photoperiodic regulation of tph2 expression. Europ J Neurosc 2011;33:1308-17. prospects. J Biol Rhythms 2005;20:291-303. Anatomical demonstration of the suprachiasmatic nucleus- pineal pathway. J Comp Neurol 1999;406:171-82. Suprachiasmatic control of melatonin synthesis in rats: inhibitory and stimulatory mechanisms. Eur J Neurosci 2003;17:221-8. evidence for a controlled offset of melatonin synthesis at dawn by the suprachiasmatic nucleus in the rat. Neuroscience 2005;130:797-803. Neuroendocrinol 2003;15:1-5. attempt to correlate brain areas containing melatonin binding sites with rhythmic functions: A study in five hibernator species. Cell Tiss Res 1994;278:97-106. Neurosciences 2002;4:57-72. specific abolition of Per1 expression in the pars tuberalis by pinealectomy in the Syrian hamster. Neuroreport 2001;12:579-82. induces Cry1 expression in the pars tuberalis of the rat. Mol Brain Res 2003;114:91-182. circadian clock entrained by melatonin is ticking in the rat fetal adrenal. Endocrinology 2011;152:1891-900. Med Rev 2005;9:25-39. effect of melatonin. Cell Tiss Res 2002;309:183-191. circadian rhythms during daily infusion of melatonin or S20098, a melatonin agonist. Am J Physiol 1999;277:R8126-R828. dose 0.5-mg melatonin on the free-running circadian rhythms of blind subjects. J Biol Rhythms 2003;18:420-9. confirmatory result using a simplified protocol. Biol Psychiatry 1992;32:705-6. elderly. Exp Gerontol 2000;35:1229-37. relevance. Exp Gerontol 2001;36:1083-100. sclérose tubéreuse de Bourneville mise à jour des connais- gine génétique et d'expression surtout neuro- logique et psychiatrique. Des avancées géné- tiques et biochimiques permettent d'envisa- ger des progrès manifestes dans la prise en charge. Il est donc important d'éviter le «sous-diagnostic» et le diagnostic tardif. pour les médecins? de la «clause de non-concurrence». En vertu peut exercer aucune activité similaire. Autrement dit, ce collabo- rateur ne peut ni lancer lui-même une société, ni exercer dans une entreprise concurrente de son ex-employeur une fonction risquant de porter préjudice à ce dernier. L'ex-collabora- teur en question pourrait notam- ment utiliser à des fins personnelles ou à l'avantage d'une en- treprise concurrente les connaissances acquises dans la société de son ex-employeur. |